The Role of Drugs in AIDS

1/10/93 Ms. No. T1867

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widespread acceptance of the virus-AIDS hypothesis and the failure to
consider the enormous difference between the viral and cellular targets.
Conclusions
It is concluded that most or all American AIDS, that exceeds the
normal low incidence of indicator diseases in the general population, is the
result of recreational and anti-HIV drugs, and that the American AIDS
epidemic is that subset of the drug epidemic, which is confirmed or assumed
HIV-positive. For example, only the pneumonias, tuberculoses, and
dementias of the 50% of American intravenous drug users with HIV (7) will
be recorded as AIDS.
It would appear that the drug-AIDS hypothesis is epidemiologically
and pathologically better grounded than the virus-AIDS hypothesis. About
32% American AIDS patients are confirmed intravenous drug users, possibly
60% use recreational drugs orally and an unknown but large percentage of
both behavioral and clinical AIDS risk groups use AZT. The consumption of
recreational drugs by AIDS patients is probably underreported, because the
drugs are illicit and because medical scientists and support for research are
currently heavily biased in favor of viral AIDS (27, 92, 113). By contrast
antibodies to HIV are confirmed in only about 50% of AIDS patients (6, 19).
Furthermore, the pathogenicity of drugs consumed by AIDS patients is
empirically known for all, and mechanistically for some drugs, notably for
AZT and nitrites. But it is a complete mystery how HIV acts as a pathogen,
despite enormous research efforts (3, 114-116).
The drug-AIDS hypothesis resolves all scientific paradoxes posed by the
prevailing virus-AIDS hypothesis:
1) The AIDS epidemic is new but the HIV epidemic is old in Americabecause the drug epidemic is new.