Cranium (a) has an empty RP4 alveolus and a loose calotte. Individual suffered from osteitis deformans (Paget's disease). The LI1-2 and RI1 alveoli are resorbed, and most teeth worn down to rounded peg-like structures. Postglenoid processes are prominent, there are bilateral rugosities at posterolateral maxillary hard palate, and the spheno-occipital synchondrosis is unfused or broken. There is a tubercle at basion, resorption of the iniac region, doubled right supraorbital foramina. Hypertrophied facial bone 'bridges' the over routes of the left supratrochlear and infraorbital vessels (probably related to osteitis). The right mastoid foramen is enlarged. Mandible (b) with most lower teeth present
Cranium (a) has an empty RP4 alveolus and a loose calotte. Individual suffered from osteitis deformans (Paget's disease). The LI1-2 and RI1 alveoli are resorbed, and most teeth worn down to rounded peg-like structures. Postglenoid processes are prominent, there are bilateral rugosities at posterolateral maxillary hard palate, and the spheno-occipital synchondrosis is unfused or broken. There is a tubercle at basion, resorption of the iniac region, doubled right supraorbital foramina. Hypertrophied facial bone 'bridges' the over routes of the left supratrochlear and infraorbital vessels (probably related to osteitis). The right mastoid foramen is enlarged. Mandible (b) with most lower teeth present
Cranium (a) has an empty RP4 alveolus and a loose calotte. Individual suffered from osteitis deformans (Paget's disease). The LI1-2 and RI1 alveoli are resorbed, and most teeth worn down to rounded peg-like structures. Postglenoid processes are prominent, there are bilateral rugosities at posterolateral maxillary hard palate, and the spheno-occipital synchondrosis is unfused or broken. There is a tubercle at basion, resorption of the iniac region, doubled right supraorbital foramina. Hypertrophied facial bone 'bridges' the over routes of the left supratrochlear and infraorbital vessels (probably related to osteitis). The right mastoid foramen is enlarged. Mandible (b) with most lower teeth present
Cranium (a) has an empty RP4 alveolus and a loose calotte. Individual suffered from osteitis deformans (Paget's disease). The LI1-2 and RI1 alveoli are resorbed, and most teeth worn down to rounded peg-like structures. Postglenoid processes are prominent, there are bilateral rugosities at posterolateral maxillary hard palate, and the spheno-occipital synchondrosis is unfused or broken. There is a tubercle at basion, resorption of the iniac region, doubled right supraorbital foramina. Hypertrophied facial bone 'bridges' the over routes of the left supratrochlear and infraorbital vessels (probably related to osteitis). The right mastoid foramen is enlarged. Mandible (b) with most lower teeth present
Cranium (a) has an empty RP4 alveolus and a loose calotte. Individual suffered from osteitis deformans (Paget's disease). The LI1-2 and RI1 alveoli are resorbed, and most teeth worn down to rounded peg-like structures. Postglenoid processes are prominent, there are bilateral rugosities at posterolateral maxillary hard palate, and the spheno-occipital synchondrosis is unfused or broken. There is a tubercle at basion, resorption of the iniac region, doubled right supraorbital foramina. Hypertrophied facial bone 'bridges' the over routes of the left supratrochlear and infraorbital vessels (probably related to osteitis). The right mastoid foramen is enlarged. Mandible (b) with most lower teeth present
Cranium (a) has an empty RP4 alveolus and a loose calotte. Individual suffered from osteitis deformans (Paget's disease). The LI1-2 and RI1 alveoli are resorbed, and most teeth worn down to rounded peg-like structures. Postglenoid processes are prominent, there are bilateral rugosities at posterolateral maxillary hard palate, and the spheno-occipital synchondrosis is unfused or broken. There is a tubercle at basion, resorption of the iniac region, doubled right supraorbital foramina. Hypertrophied facial bone 'bridges' the over routes of the left supratrochlear and infraorbital vessels (probably related to osteitis). The right mastoid foramen is enlarged. Mandible (b) with most lower teeth present
Cranium (a) has an empty RP4 alveolus and a loose calotte. Individual suffered from osteitis deformans (Paget's disease). The LI1-2 and RI1 alveoli are resorbed, and most teeth worn down to rounded peg-like structures. Postglenoid processes are prominent, there are bilateral rugosities at posterolateral maxillary hard palate, and the spheno-occipital synchondrosis is unfused or broken. There is a tubercle at basion, resorption of the iniac region, doubled right supraorbital foramina. Hypertrophied facial bone 'bridges' the over routes of the left supratrochlear and infraorbital vessels (probably related to osteitis). The right mastoid foramen is enlarged. Mandible (b) with most lower teeth present
Cranium (a) has an empty RP4 alveolus and a loose calotte. Individual suffered from osteitis deformans (Paget's disease). The LI1-2 and RI1 alveoli are resorbed, and most teeth worn down to rounded peg-like structures. Postglenoid processes are prominent, there are bilateral rugosities at posterolateral maxillary hard palate, and the spheno-occipital synchondrosis is unfused or broken. There is a tubercle at basion, resorption of the iniac region, doubled right supraorbital foramina. Hypertrophied facial bone 'bridges' the over routes of the left supratrochlear and infraorbital vessels (probably related to osteitis). The right mastoid foramen is enlarged. Mandible (b) with most lower teeth present
Cranium (a) has an empty RP4 alveolus and a loose calotte. Individual suffered from osteitis deformans (Paget's disease). The LI1-2 and RI1 alveoli are resorbed, and most teeth worn down to rounded peg-like structures. Postglenoid processes are prominent, there are bilateral rugosities at posterolateral maxillary hard palate, and the spheno-occipital synchondrosis is unfused or broken. There is a tubercle at basion, resorption of the iniac region, doubled right supraorbital foramina. Hypertrophied facial bone 'bridges' the over routes of the left supratrochlear and infraorbital vessels (probably related to osteitis). The right mastoid foramen is enlarged. Mandible (b) with most lower teeth present
Cranium (a) has an empty RP4 alveolus and a loose calotte. Individual suffered from osteitis deformans (Paget's disease). The LI1-2 and RI1 alveoli are resorbed, and most teeth worn down to rounded peg-like structures. Postglenoid processes are prominent, there are bilateral rugosities at posterolateral maxillary hard palate, and the spheno-occipital synchondrosis is unfused or broken. There is a tubercle at basion, resorption of the iniac region, doubled right supraorbital foramina. Hypertrophied facial bone 'bridges' the over routes of the left supratrochlear and infraorbital vessels (probably related to osteitis). The right mastoid foramen is enlarged. Mandible (b) with most lower teeth present
The calotte’s cranium (a) is loose. All alveoli are resorbed. The posterior calvaria is depressed, and there are lesions endocranially. Bone is atrophied at right and left asterion. The occipital condyles and temporo-mandibular joint are flat and arthritic. All the mandible’s (b) alveoli are resorbed.
The calotte’s cranium (a) is loose. All alveoli are resorbed. The posterior calvaria is depressed, and there are lesions endocranially. Bone is atrophied at right and left asterion. The occipital condyles and temporo-mandibular joint are flat and arthritic. All the mandible’s (b) alveoli are resorbed.
The calotte’s cranium (a) is loose. All alveoli are resorbed. The posterior calvaria is depressed, and there are lesions endocranially. Bone is atrophied at right and left asterion. The occipital condyles and temporo-mandibular joint are flat and arthritic. All the mandible’s (b) alveoli are resorbed.
The calotte’s cranium (a) is loose. All alveoli are resorbed. The posterior calvaria is depressed, and there are lesions endocranially. Bone is atrophied at right and left asterion. The occipital condyles and temporo-mandibular joint are flat and arthritic. All the mandible’s (b) alveoli are resorbed.
The calotte’s cranium (a) is loose. All alveoli are resorbed. The posterior calvaria is depressed, and there are lesions endocranially. Bone is atrophied at right and left asterion. The occipital condyles and temporo-mandibular joint are flat and arthritic. All the mandible’s (b) alveoli are resorbed.
The calotte’s cranium (a) is loose. All alveoli are resorbed. The posterior calvaria is depressed, and there are lesions endocranially. Bone is atrophied at right and left asterion. The occipital condyles and temporo-mandibular joint are flat and arthritic. All the mandible’s (b) alveoli are resorbed.
The calotte’s cranium (a) is loose. All alveoli are resorbed. The posterior calvaria is depressed, and there are lesions endocranially. Bone is atrophied at right and left asterion. The occipital condyles and temporo-mandibular joint are flat and arthritic. All the mandible’s (b) alveoli are resorbed.
The calotte’s cranium (a) is loose. All alveoli are resorbed. The posterior calvaria is depressed, and there are lesions endocranially. Bone is atrophied at right and left asterion. The occipital condyles and temporo-mandibular joint are flat and arthritic. All the mandible’s (b) alveoli are resorbed.
The calotte’s cranium (a) is loose. All alveoli are resorbed. The posterior calvaria is depressed, and there are lesions endocranially. Bone is atrophied at right and left asterion. The occipital condyles and temporo-mandibular joint are flat and arthritic. All the mandible’s (b) alveoli are resorbed.
The calotte’s cranium (a) is loose. All alveoli are resorbed. The posterior calvaria is depressed, and there are lesions endocranially. Bone is atrophied at right and left asterion. The occipital condyles and temporo-mandibular joint are flat and arthritic. All the mandible’s (b) alveoli are resorbed.
The calotte’s cranium (a) is loose. All alveoli are resorbed. The posterior calvaria is depressed, and there are lesions endocranially. Bone is atrophied at right and left asterion. The occipital condyles and temporo-mandibular joint are flat and arthritic. All the mandible’s (b) alveoli are resorbed.
The calotte’s cranium (a) is loose. All alveoli are resorbed. The posterior calvaria is depressed, and there are lesions endocranially. Bone is atrophied at right and left asterion. The occipital condyles and temporo-mandibular joint are flat and arthritic. All the mandible’s (b) alveoli are resorbed.
The calotte’s cranium (a) is loose. All alveoli are resorbed. The posterior calvaria is depressed, and there are lesions endocranially. Bone is atrophied at right and left asterion. The occipital condyles and temporo-mandibular joint are flat and arthritic. All the mandible’s (b) alveoli are resorbed.
The calotte’s cranium (a) is loose. All alveoli are resorbed. The posterior calvaria is depressed, and there are lesions endocranially. Bone is atrophied at right and left asterion. The occipital condyles and temporo-mandibular joint are flat and arthritic. All the mandible’s (b) alveoli are resorbed.
The calotte’s cranium (a) is loose. All alveoli are resorbed. The posterior calvaria is depressed, and there are lesions endocranially. Bone is atrophied at right and left asterion. The occipital condyles and temporo-mandibular joint are flat and arthritic. All the mandible’s (b) alveoli are resorbed.
The calotte’s cranium (a) is loose. All alveoli are resorbed. The posterior calvaria is depressed, and there are lesions endocranially. Bone is atrophied at right and left asterion. The occipital condyles and temporo-mandibular joint are flat and arthritic. All the mandible’s (b) alveoli are resorbed.
The calotte’s cranium (a) is loose. All alveoli are resorbed. The posterior calvaria is depressed, and there are lesions endocranially. Bone is atrophied at right and left asterion. The occipital condyles and temporo-mandibular joint are flat and arthritic. All the mandible’s (b) alveoli are resorbed.
The calotte’s cranium (a) is loose. All alveoli are resorbed. The posterior calvaria is depressed, and there are lesions endocranially. Bone is atrophied at right and left asterion. The occipital condyles and temporo-mandibular joint are flat and arthritic. All the mandible’s (b) alveoli are resorbed.
The calotte’s cranium (a) is loose. All alveoli are resorbed. The posterior calvaria is depressed, and there are lesions endocranially. Bone is atrophied at right and left asterion. The occipital condyles and temporo-mandibular joint are flat and arthritic. All the mandible’s (b) alveoli are resorbed.
The calotte’s cranium (a) is loose. All alveoli are resorbed. The posterior calvaria is depressed, and there are lesions endocranially. Bone is atrophied at right and left asterion. The occipital condyles and temporo-mandibular joint are flat and arthritic. All the mandible’s (b) alveoli are resorbed.
The calotte’s cranium (a) is loose. All alveoli are resorbed. The posterior calvaria is depressed, and there are lesions endocranially. Bone is atrophied at right and left asterion. The occipital condyles and temporo-mandibular joint are flat and arthritic. All the mandible’s (b) alveoli are resorbed.
Cranium (a) is missing the calotte, most whole teeth or crowns are missing (only the LM2 is intact), and there is some alveolar resorption. There is a bony build-up on dorsum sellae resulting in a deeply excavated pituitary fossa. Lesions mark the middle cranial fossae resulting in a hole in the anterosuperior lateral corner of L greater sphenoid wing. The middle meningeal arteries are deeply excavated. The sphenoid greater wings appear hypertrophied, as does the globular crista galli. The mastoid foramina are very large and open visibly into sigmoid sinuses (viz. on the right). The left foramen spinosum is bridged ectocranially, the lambdoidal suture is almost completely obliterated. The vault may have been expanded relative to face, though it is hard to say without calotte. Mandible (b) retains all molars but some crowns are broken, and lacks the other teeth. There are silver fillings on occlusal surfaces of all molars but RM2. The empty canine alveoli show they were each rotated distally. The lingulae are blunt, rather than pointed.
Cranium (a) is missing the calotte, most whole teeth or crowns are missing (only the LM2 is intact), and there is some alveolar resorption. There is a bony build-up on dorsum sellae resulting in a deeply excavated pituitary fossa. Lesions mark the middle cranial fossae resulting in a hole in the anterosuperior lateral corner of L greater sphenoid wing. The middle meningeal arteries are deeply excavated. The sphenoid greater wings appear hypertrophied, as does the globular crista galli. The mastoid foramina are very large and open visibly into sigmoid sinuses (viz. on the right). The left foramen spinosum is bridged ectocranially, the lambdoidal suture is almost completely obliterated. The vault may have been expanded relative to face, though it is hard to say without calotte. Mandible (b) retains all molars but some crowns are broken, and lacks the other teeth. There are silver fillings on occlusal surfaces of all molars but RM2. The empty canine alveoli show they were each rotated distally. The lingulae are blunt, rather than pointed.
Cranium (a) is missing the calotte, most whole teeth or crowns are missing (only the LM2 is intact), and there is some alveolar resorption. There is a bony build-up on dorsum sellae resulting in a deeply excavated pituitary fossa. Lesions mark the middle cranial fossae resulting in a hole in the anterosuperior lateral corner of L greater sphenoid wing. The middle meningeal arteries are deeply excavated. The sphenoid greater wings appear hypertrophied, as does the globular crista galli. The mastoid foramina are very large and open visibly into sigmoid sinuses (viz. on the right). The left foramen spinosum is bridged ectocranially, the lambdoidal suture is almost completely obliterated. The vault may have been expanded relative to face, though it is hard to say without calotte. Mandible (b) retains all molars but some crowns are broken, and lacks the other teeth. There are silver fillings on occlusal surfaces of all molars but RM2. The empty canine alveoli show they were each rotated distally. The lingulae are blunt, rather than pointed.
Cranium (a) is missing the calotte, most whole teeth or crowns are missing (only the LM2 is intact), and there is some alveolar resorption. There is a bony build-up on dorsum sellae resulting in a deeply excavated pituitary fossa. Lesions mark the middle cranial fossae resulting in a hole in the anterosuperior lateral corner of L greater sphenoid wing. The middle meningeal arteries are deeply excavated. The sphenoid greater wings appear hypertrophied, as does the globular crista galli. The mastoid foramina are very large and open visibly into sigmoid sinuses (viz. on the right). The left foramen spinosum is bridged ectocranially, the lambdoidal suture is almost completely obliterated. The vault may have been expanded relative to face, though it is hard to say without calotte. Mandible (b) retains all molars but some crowns are broken, and lacks the other teeth. There are silver fillings on occlusal surfaces of all molars but RM2. The empty canine alveoli show they were each rotated distally. The lingulae are blunt, rather than pointed.
Cranium (a) is missing the calotte, most whole teeth or crowns are missing (only the LM2 is intact), and there is some alveolar resorption. There is a bony build-up on dorsum sellae resulting in a deeply excavated pituitary fossa. Lesions mark the middle cranial fossae resulting in a hole in the anterosuperior lateral corner of L greater sphenoid wing. The middle meningeal arteries are deeply excavated. The sphenoid greater wings appear hypertrophied, as does the globular crista galli. The mastoid foramina are very large and open visibly into sigmoid sinuses (viz. on the right). The left foramen spinosum is bridged ectocranially, the lambdoidal suture is almost completely obliterated. The vault may have been expanded relative to face, though it is hard to say without calotte. Mandible (b) retains all molars but some crowns are broken, and lacks the other teeth. There are silver fillings on occlusal surfaces of all molars but RM2. The empty canine alveoli show they were each rotated distally. The lingulae are blunt, rather than pointed.
Cranium (a) is missing the calotte, most whole teeth or crowns are missing (only the LM2 is intact), and there is some alveolar resorption. There is a bony build-up on dorsum sellae resulting in a deeply excavated pituitary fossa. Lesions mark the middle cranial fossae resulting in a hole in the anterosuperior lateral corner of L greater sphenoid wing. The middle meningeal arteries are deeply excavated. The sphenoid greater wings appear hypertrophied, as does the globular crista galli. The mastoid foramina are very large and open visibly into sigmoid sinuses (viz. on the right). The left foramen spinosum is bridged ectocranially, the lambdoidal suture is almost completely obliterated. The vault may have been expanded relative to face, though it is hard to say without calotte. Mandible (b) retains all molars but some crowns are broken, and lacks the other teeth. There are silver fillings on occlusal surfaces of all molars but RM2. The empty canine alveoli show they were each rotated distally. The lingulae are blunt, rather than pointed.
Cranium (a) is missing the calotte, most whole teeth or crowns are missing (only the LM2 is intact), and there is some alveolar resorption. There is a bony build-up on dorsum sellae resulting in a deeply excavated pituitary fossa. Lesions mark the middle cranial fossae resulting in a hole in the anterosuperior lateral corner of L greater sphenoid wing. The middle meningeal arteries are deeply excavated. The sphenoid greater wings appear hypertrophied, as does the globular crista galli. The mastoid foramina are very large and open visibly into sigmoid sinuses (viz. on the right). The left foramen spinosum is bridged ectocranially, the lambdoidal suture is almost completely obliterated. The vault may have been expanded relative to face, though it is hard to say without calotte. Mandible (b) retains all molars but some crowns are broken, and lacks the other teeth. There are silver fillings on occlusal surfaces of all molars but RM2. The empty canine alveoli show they were each rotated distally. The lingulae are blunt, rather than pointed.
Cranium (a) is missing the calotte, most whole teeth or crowns are missing (only the LM2 is intact), and there is some alveolar resorption. There is a bony build-up on dorsum sellae resulting in a deeply excavated pituitary fossa. Lesions mark the middle cranial fossae resulting in a hole in the anterosuperior lateral corner of L greater sphenoid wing. The middle meningeal arteries are deeply excavated. The sphenoid greater wings appear hypertrophied, as does the globular crista galli. The mastoid foramina are very large and open visibly into sigmoid sinuses (viz. on the right). The left foramen spinosum is bridged ectocranially, the lambdoidal suture is almost completely obliterated. The vault may have been expanded relative to face, though it is hard to say without calotte. Mandible (b) retains all molars but some crowns are broken, and lacks the other teeth. There are silver fillings on occlusal surfaces of all molars but RM2. The empty canine alveoli show they were each rotated distally. The lingulae are blunt, rather than pointed.
Cranium (a) is missing the calotte, most whole teeth or crowns are missing (only the LM2 is intact), and there is some alveolar resorption. There is a bony build-up on dorsum sellae resulting in a deeply excavated pituitary fossa. Lesions mark the middle cranial fossae resulting in a hole in the anterosuperior lateral corner of L greater sphenoid wing. The middle meningeal arteries are deeply excavated. The sphenoid greater wings appear hypertrophied, as does the globular crista galli. The mastoid foramina are very large and open visibly into sigmoid sinuses (viz. on the right). The left foramen spinosum is bridged ectocranially, the lambdoidal suture is almost completely obliterated. The vault may have been expanded relative to face, though it is hard to say without calotte. Mandible (b) retains all molars but some crowns are broken, and lacks the other teeth. There are silver fillings on occlusal surfaces of all molars but RM2. The empty canine alveoli show they were each rotated distally. The lingulae are blunt, rather than pointed.
Cranium (a) is missing the calotte, most whole teeth or crowns are missing (only the LM2 is intact), and there is some alveolar resorption. There is a bony build-up on dorsum sellae resulting in a deeply excavated pituitary fossa. Lesions mark the middle cranial fossae resulting in a hole in the anterosuperior lateral corner of L greater sphenoid wing. The middle meningeal arteries are deeply excavated. The sphenoid greater wings appear hypertrophied, as does the globular crista galli. The mastoid foramina are very large and open visibly into sigmoid sinuses (viz. on the right). The left foramen spinosum is bridged ectocranially, the lambdoidal suture is almost completely obliterated. The vault may have been expanded relative to face, though it is hard to say without calotte. Mandible (b) retains all molars but some crowns are broken, and lacks the other teeth. There are silver fillings on occlusal surfaces of all molars but RM2. The empty canine alveoli show they were each rotated distally. The lingulae are blunt, rather than pointed.
Cranium (a) is missing the calotte, most whole teeth or crowns are missing (only the LM2 is intact), and there is some alveolar resorption. There is a bony build-up on dorsum sellae resulting in a deeply excavated pituitary fossa. Lesions mark the middle cranial fossae resulting in a hole in the anterosuperior lateral corner of L greater sphenoid wing. The middle meningeal arteries are deeply excavated. The sphenoid greater wings appear hypertrophied, as does the globular crista galli. The mastoid foramina are very large and open visibly into sigmoid sinuses (viz. on the right). The left foramen spinosum is bridged ectocranially, the lambdoidal suture is almost completely obliterated. The vault may have been expanded relative to face, though it is hard to say without calotte. Mandible (b) retains all molars but some crowns are broken, and lacks the other teeth. There are silver fillings on occlusal surfaces of all molars but RM2. The empty canine alveoli show they were each rotated distally. The lingulae are blunt, rather than pointed.
Cranium (a) is missing the calotte, most whole teeth or crowns are missing (only the LM2 is intact), and there is some alveolar resorption. There is a bony build-up on dorsum sellae resulting in a deeply excavated pituitary fossa. Lesions mark the middle cranial fossae resulting in a hole in the anterosuperior lateral corner of L greater sphenoid wing. The middle meningeal arteries are deeply excavated. The sphenoid greater wings appear hypertrophied, as does the globular crista galli. The mastoid foramina are very large and open visibly into sigmoid sinuses (viz. on the right). The left foramen spinosum is bridged ectocranially, the lambdoidal suture is almost completely obliterated. The vault may have been expanded relative to face, though it is hard to say without calotte. Mandible (b) retains all molars but some crowns are broken, and lacks the other teeth. There are silver fillings on occlusal surfaces of all molars but RM2. The empty canine alveoli show they were each rotated distally. The lingulae are blunt, rather than pointed.
Cranium (a) is missing the calotte, most whole teeth or crowns are missing (only the LM2 is intact), and there is some alveolar resorption. There is a bony build-up on dorsum sellae resulting in a deeply excavated pituitary fossa. Lesions mark the middle cranial fossae resulting in a hole in the anterosuperior lateral corner of L greater sphenoid wing. The middle meningeal arteries are deeply excavated. The sphenoid greater wings appear hypertrophied, as does the globular crista galli. The mastoid foramina are very large and open visibly into sigmoid sinuses (viz. on the right). The left foramen spinosum is bridged ectocranially, the lambdoidal suture is almost completely obliterated. The vault may have been expanded relative to face, though it is hard to say without calotte. Mandible (b) retains all molars but some crowns are broken, and lacks the other teeth. There are silver fillings on occlusal surfaces of all molars but RM2. The empty canine alveoli show they were each rotated distally. The lingulae are blunt, rather than pointed.
Cranium (a) is missing the calotte, most whole teeth or crowns are missing (only the LM2 is intact), and there is some alveolar resorption. There is a bony build-up on dorsum sellae resulting in a deeply excavated pituitary fossa. Lesions mark the middle cranial fossae resulting in a hole in the anterosuperior lateral corner of L greater sphenoid wing. The middle meningeal arteries are deeply excavated. The sphenoid greater wings appear hypertrophied, as does the globular crista galli. The mastoid foramina are very large and open visibly into sigmoid sinuses (viz. on the right). The left foramen spinosum is bridged ectocranially, the lambdoidal suture is almost completely obliterated. The vault may have been expanded relative to face, though it is hard to say without calotte. Mandible (b) retains all molars but some crowns are broken, and lacks the other teeth. There are silver fillings on occlusal surfaces of all molars but RM2. The empty canine alveoli show they were each rotated distally. The lingulae are blunt, rather than pointed.
Cranium (a) is missing the calotte, most whole teeth or crowns are missing (only the LM2 is intact), and there is some alveolar resorption. There is a bony build-up on dorsum sellae resulting in a deeply excavated pituitary fossa. Lesions mark the middle cranial fossae resulting in a hole in the anterosuperior lateral corner of L greater sphenoid wing. The middle meningeal arteries are deeply excavated. The sphenoid greater wings appear hypertrophied, as does the globular crista galli. The mastoid foramina are very large and open visibly into sigmoid sinuses (viz. on the right). The left foramen spinosum is bridged ectocranially, the lambdoidal suture is almost completely obliterated. The vault may have been expanded relative to face, though it is hard to say without calotte. Mandible (b) retains all molars but some crowns are broken, and lacks the other teeth. There are silver fillings on occlusal surfaces of all molars but RM2. The empty canine alveoli show they were each rotated distally. The lingulae are blunt, rather than pointed.
Cranium (a) is missing the calotte, most whole teeth or crowns are missing (only the LM2 is intact), and there is some alveolar resorption. There is a bony build-up on dorsum sellae resulting in a deeply excavated pituitary fossa. Lesions mark the middle cranial fossae resulting in a hole in the anterosuperior lateral corner of L greater sphenoid wing. The middle meningeal arteries are deeply excavated. The sphenoid greater wings appear hypertrophied, as does the globular crista galli. The mastoid foramina are very large and open visibly into sigmoid sinuses (viz. on the right). The left foramen spinosum is bridged ectocranially, the lambdoidal suture is almost completely obliterated. The vault may have been expanded relative to face, though it is hard to say without calotte. Mandible (b) retains all molars but some crowns are broken, and lacks the other teeth. There are silver fillings on occlusal surfaces of all molars but RM2. The empty canine alveoli show they were each rotated distally. The lingulae are blunt, rather than pointed.
Cranium (a) is missing the calotte, most whole teeth or crowns are missing (only the LM2 is intact), and there is some alveolar resorption. There is a bony build-up on dorsum sellae resulting in a deeply excavated pituitary fossa. Lesions mark the middle cranial fossae resulting in a hole in the anterosuperior lateral corner of L greater sphenoid wing. The middle meningeal arteries are deeply excavated. The sphenoid greater wings appear hypertrophied, as does the globular crista galli. The mastoid foramina are very large and open visibly into sigmoid sinuses (viz. on the right). The left foramen spinosum is bridged ectocranially, the lambdoidal suture is almost completely obliterated. The vault may have been expanded relative to face, though it is hard to say without calotte. Mandible (b) retains all molars but some crowns are broken, and lacks the other teeth. There are silver fillings on occlusal surfaces of all molars but RM2. The empty canine alveoli show they were each rotated distally. The lingulae are blunt, rather than pointed.
Cranium (a) is missing the calotte, most whole teeth or crowns are missing (only the LM2 is intact), and there is some alveolar resorption. There is a bony build-up on dorsum sellae resulting in a deeply excavated pituitary fossa. Lesions mark the middle cranial fossae resulting in a hole in the anterosuperior lateral corner of L greater sphenoid wing. The middle meningeal arteries are deeply excavated. The sphenoid greater wings appear hypertrophied, as does the globular crista galli. The mastoid foramina are very large and open visibly into sigmoid sinuses (viz. on the right). The left foramen spinosum is bridged ectocranially, the lambdoidal suture is almost completely obliterated. The vault may have been expanded relative to face, though it is hard to say without calotte. Mandible (b) retains all molars but some crowns are broken, and lacks the other teeth. There are silver fillings on occlusal surfaces of all molars but RM2. The empty canine alveoli show they were each rotated distally. The lingulae are blunt, rather than pointed.
Cranium (a) is missing the calotte, most whole teeth or crowns are missing (only the LM2 is intact), and there is some alveolar resorption. There is a bony build-up on dorsum sellae resulting in a deeply excavated pituitary fossa. Lesions mark the middle cranial fossae resulting in a hole in the anterosuperior lateral corner of L greater sphenoid wing. The middle meningeal arteries are deeply excavated. The sphenoid greater wings appear hypertrophied, as does the globular crista galli. The mastoid foramina are very large and open visibly into sigmoid sinuses (viz. on the right). The left foramen spinosum is bridged ectocranially, the lambdoidal suture is almost completely obliterated. The vault may have been expanded relative to face, though it is hard to say without calotte. Mandible (b) retains all molars but some crowns are broken, and lacks the other teeth. There are silver fillings on occlusal surfaces of all molars but RM2. The empty canine alveoli show they were each rotated distally. The lingulae are blunt, rather than pointed.
